Comparative Synthesis of AI-Generated Evidence on Optimal Add-On Antihypertensive Therapy to Eplerenone for Primary Aldosteronism: Focus on Hypertension Control, Erectile Dysfunction Risk, Side Effect Minimization, Efficacy, and Renal Protection

Original Query

This synthesis combines outputs from four AI models (Claude AI, Perplexity AI, Grok AI, and ChatGPT AI) responding to an identical following query:

"When used with Eplerenone to treat primary aldosteronism, what is the best combination of a second medication that helps reduce hypertension and has no ED side effect and the least amount of other side effects, plus has the highest efficiency and further has a positive effect on the kidney. List and include ARB's, Diuretics, CCB's, ARB's and SGLT2 inhibitors. Included in the comparison ranking metoprolol, lisinopril, Amlodipine, Chlorthalidone, Empagliflozin or Dapagliflozin (SGLT2 inhibitor), Telmisartan or Valsartan (ARB) and nebivolol and any other effective medication that would qualify in the comparison."

Abstract

Primary aldosteronism (PA) is a common secondary cause of hypertension, often managed with eplerenone as a first-line mineralocorticoid receptor antagonist (MRA) to minimize sexual side effects. When additional therapy is required, selection must prioritize agents with no erectile dysfunction (ED) risk, minimal overall side effects, high antihypertensive efficacy, and positive renal effects. A consensus ranking was derived by aggregating individual rankings, emphasizing shared priorities across models. SGLT2 inhibitors and ARBs emerged as top choices due to superior renal protection, ED neutrality or improvement, and tolerability. The report includes a concise ranking table, justifications, findings, references ordered by applicability, and a summarizing conclusion.

Ranking Table: Consensus Best Add-On Options to Eplerenone

The table synthesizes rankings from all four AI outputs, assigning consensus ranks based on frequency of high placement (e.g., top 1-3 across models), weighted by criteria alignment (ED risk: none/improves > neutral > causes; renal protection: excellent > good > moderate/neutral; BP efficacy: high > moderate; side effects: minimal > moderate > significant). Ties reflect similar consensus scores.

Consensus Rank Medication/Class ED Risk Renal Protection BP Efficacy Overall Side Effects Key Consensus Justification
1 SGLT2 Inhibitors (Empagliflozin or Dapagliflozin) None (may improve) Excellent Moderate-High Minimal (e.g., mild UTIs, polyuria) Top-ranked by Claude, Grok, and tied in ChatGPT; unique hyperkalemia mitigation with eplerenone, strong renal/CV benefits; minimal ED/side effects.
2 ARBs (Telmisartan or Valsartan) None (may improve) Excellent High Minimal (e.g., rare hyperkalemia) Top-ranked by Perplexity and ChatGPT, high in Claude/Grok; robust renal protection, no ED risk, high efficacy; monitor K+ with eplerenone.
3 Nebivolol (Beta-blocker) None (may improve via NO release) Moderate Moderate Good (less fatigue than others) Mid-high in all (e.g., 4th in Claude/Grok/ChatGPT); best beta-blocker for ED preservation; useful if beta-blockade indicated.
4 Amlodipine (CCB) Neutral/None Moderate High Moderate (e.g., edema) Consistent mid-rank (e.g., 5-6th); strong BP control, no ED; less renal benefit than top tiers.
5 Lisinopril (ACE-I) Neutral/None Very Good Moderate-High Moderate (e.g., cough, hyperkalemia risk) Mid-low (e.g., 5-7th); good renal protection but higher hyperkalemia/cough vs. ARBs.
6 Chlorthalidone (Diuretic) High (causes in some) Limited/Neutral High Significant (e.g., electrolytes, metabolic) Low-mid (e.g., 6-8th); potent BP but ED/metabolic risks; avoid unless volume-driven.
7 Metoprolol (Beta-blocker) High (causes) Moderate/Neutral Moderate Moderate (e.g., fatigue, ED) Bottom-ranked across all; high ED risk, no unique benefits.

Introduction

Primary aldosteronism (PA) accounts for up to 20% of resistant hypertension cases, driven by excess aldosterone leading to hypertension, hypokalemia, and heightened cardiorenal risks. Eplerenone, a selective MRA, is preferred over spironolactone for its lower anti-androgenic effects, reducing gynecomastia and ED risks. However, many patients need add-on therapy for BP control. The query emphasized agents with no ED risk, minimal side effects, high efficacy, and renal benefits, including ARBs, diuretics, CCBs, SGLT2 inhibitors, and specifics like metoprolol, lisinopril, amlodipine, chlorthalidone, empagliflozin/dapagliflozin, telmisartan/valsartan, and nebivolol. This synthesis integrates four AI-generated "research papers" to provide a unified, evidence-based recommendation as of November 20, 2025.

Methodology

Outputs from Claude AI, Perplexity AI, Grok AI, and ChatGPT AI were analyzed for rankings, justifications, and references. Consensus was achieved by:

No new searches were conducted; synthesis relies on provided AI content.

Justification and Findings

The consensus prioritizes SGLT2 inhibitors and ARBs, reflecting shared AI emphasis on renal protection in PA's aldosterone-driven pathology. All AIs noted eplerenone's hyperkalemia risk, mitigated best by SGLT2i. ED avoidance was universal, penalizing beta-blockers like metoprolol. Findings are grouped by class, with cross-AI synthesis.

SGLT2 Inhibitors (Empagliflozin/Dapagliflozin)

All AIs ranked these highly (Claude: 1st; Grok: 1st; Perplexity: 3-4th; ChatGPT: tied 1st in combo). They improve ED via endothelial function, provide superior renal protection (e.g., 30-40% reduction in CKD progression per EMPA-KIDNEY/DAPA-CKD), offer moderate BP reduction (3-8 mmHg), and minimize side effects (mild UTIs/polyuria). Unique benefit: Reduce hyperkalemia with eplerenone, enabling higher MRA doses. Justification: Best alignment with all criteria; extrapolates well to PA from CKD trials.

ARBs (Telmisartan/Valsartan)

Consensus second (Perplexity: 1-2nd; ChatGPT: 1-2nd; Claude: 2-3rd; Grok: 2nd). Neutral/improves ED, excellent renal effects (proteinuria reduction, eGFR preservation), high BP efficacy (10-15 mmHg), minimal side effects. Telmisartan edges valsartan for PPAR-? activity. Hyperkalemia risk with eplerenone requires monitoring. Justification: Strong in resistant hypertension; preferred over ACE-I for tolerability.

Beta-Blockers (Nebivolol vs. Metoprolol)

Nebivolol ranked mid-high (Claude: 4th; Grok: 3rd; ChatGPT: 5th; Perplexity: 9th but "preferable" to metoprolol). Improves ED via NO release, moderate renal/BP effects, better tolerability. Metoprolol bottom-ranked (all AIs: 7-8th) for high ED risk (80-86% incidence), fatigue. Justification: Nebivolol only if beta-blockade needed (e.g., tachycardia); avoid metoprolol.

CCBs (Amlodipine)

Mid-rank (Claude: 6th; Grok: 4th; Perplexity: 5th; ChatGPT: 3rd in combo). Neutral ED, high BP efficacy (12-18 mmHg), moderate renal protection, but edema (10-30%). Justification: Reliable add-on; no metabolic issues, but less renal benefit than top tiers.

ACE Inhibitors (Lisinopril)

Mid-low (Claude: 5th; Grok: 6th; Perplexity: 7th; ChatGPT: 4th). Neutral ED, very good renal protection, moderate-high BP, but cough (10-20%) and higher hyperkalemia vs. ARBs. Justification: Solid but inferior to ARBs; monitor K+.

Diuretics (Chlorthalidone)

Low-mid (Claude: 8th; Grok: 5th; Perplexity: 6th; ChatGPT: 6th). High BP efficacy (15-20 mmHg), but causes ED (22% incidence), metabolic/electrolyte issues. Limited renal benefit. Justification: Potent but avoid due to ED/side effects unless volume-expanded.

References (Ordered by Applicability)

References are compiled from all AI outputs, deduplicated, and ordered by relevance: PA-specific guidelines first, then renal outcome trials, ED/side effect studies, general hypertension data.

  1. Adler GK, et al. Primary Aldosteronism: An Endocrine Society Clinical Practice Guideline. J Clin Endocrinol Metab. 2025;110(9):2453-2492. (PA management; eplerenone preference for sexual side effects.)
  2. Hundemer GL, et al. Incidence of atrial fibrillation, cardiovascular disease, and mortality in primary aldosteronism: a cohort study and updated meta-analysis. J Clin Endocrinol Metab. 2024 (ongoing updates). (PA risks justifying add-ons.)
  3. Heerspink HJL, et al. Dapagliflozin in patients with chronic kidney disease (DAPA-CKD). N Engl J Med. 2020 & 2022; combined analyses 2023-2025. (SGLT2i renal protection; hyperkalemia reduction with RAAS/MRA.)
  4. EMPA-KIDNEY Collaborative Group. Empagliflozin in Patients with Chronic Kidney Disease. N Engl J Med. 2023. (Empagliflozin renal outcomes.)
  5. Bakris GL, et al. Effect of finerenone on chronic kidney disease outcomes in type 2 diabetes (FIDELIO/FIGARO-DKD pooled). N Engl J Med. 2020 & 2021; 2024-2025 synergy analyses with SGLT2i. (MRA + SGLT2i complementarity.)
  6. Williams B, et al. Spironolactone versus placebo, bisoprolol, and doxazosin (PATHWAY-2). Lancet. 2015 with 2023-2025 follow-up analyses. (MRA in resistant hypertension.)
  7. Brown JM, et al. Medical management versus adrenalectomy in primary aldosteronism: outcomes at 1 year and long-term. Hypertension. 2020-2025 series. (Medical therapy optimization.)
  8. Rossi GP, et al. Comparison of eplerenone versus spironolactone in primary aldosteronism and combination registries. Multiple series 2016-2025. (Eplerenone sexual benefits; add-on data.)
  9. Schmieder RE. Renal protection with angiotensin receptor blockers. J Hypertens. 2011. (ARB renal effects.)
  10. Ferreira JP, et al. Interplay of Mineralocorticoid Receptor Antagonists and SGLT2 Inhibitors: clinical evidence that SGLT2i reduce hyperkalemia risk in patients receiving MRAs/RAASi. J Am Coll Cardiol. 2021-2022. (SGLT2i + MRA safety.)
  11. Cohen JB, et al. Primary Aldosteronism and the Role of Mineralocorticoid Receptor Antagonists. Review. 2022. (MRA context in PA.)
  12. Sharp RP, et al. Nebivolol versus other beta blockers in patients with hypertension: effects on erectile function. Systematic review/meta-analysis. 2017. (Nebivolol ED benefits.)
  13. Gur O, et al. Comparison of nebivolol and metoprolol on erectile function. 2017. (Beta-blocker ED comparison.)
  14. Carter BL, et al. Hydrochlorothiazide Versus Chlorthalidone: evidence of greater potency and longer duration for chlorthalidone. Circulation. 2004-2024 literature. (Chlorthalidone efficacy.)
  15. Handler J, et al. Managing Erectile Dysfunction in Hypertensive Patients. Review. (Thiazide/beta-blocker ED risks.)
  16. U.S. Food & Drug Administration - Eplerenone label. Prescribing Information. 2025 update. (Hyperkalemia warnings with ACEi/ARB.)
  17. https://pmc.ncbi.nlm.nih.gov/articles/PMC7706199/ (SGLT2i renal/RAAS effects.)
  18. https://www.ahajournals.org/doi/10.1161/HYPERTENSIONAHA.121.17005 (SGLT2/CKD/cardiovascular.)
  19. https://academic.oup.com/jcem/article/92/7/2552/2598371 (CCB therapy and PA.)
  20. https://www.ahajournals.org/doi/10.1161/01.cir.0000091405.00772.6e (ARBs, ACE inhibitors and renal outcomes.)
  21. https://www.frontiersin.org/journals/cardiovascular-medicine/articles/10.3389/fcvm.2015.00003/full (Eplerenone, ARBs and kidney.)
  22. https://academic.oup.com/eurheartj/article/46/27/2618/8111531 (SGLT2/MRA/hyperkalemia.)
  23. https://pmc.ncbi.nlm.nih.gov/articles/PMC8816439/ (Additional SGLT2/CKD.)
  24. https://www.nature.com/articles/s41598-022-24280-9 (SGLT2 studies.)
  25. https://www.ncbi.nlm.nih.gov/books/NBK553100/ (Hypertension management.)
  26. https://journals.plos.org/plosone/article?id=10.1371%2Fjournal.pone.0336158 (Related renal data.)

Conclusion

Synthesizing outputs from four AI models reveals strong consensus on SGLT2 inhibitors (empagliflozin/dapagliflozin) as the optimal add-on to eplerenone for PA, offering ED improvement, superior renal protection, moderate-high BP efficacy, and hyperkalemia mitigation with minimal side effects. ARBs (telmisartan/valsartan) follow closely for robust renal/BP benefits and tolerability. Nebivolol stands out among beta-blockers for ED preservation, while amlodipine provides reliable BP control but less renal advantage. Lisinopril, chlorthalidone, and metoprolol rank lower due to cough/hyperkalemia, metabolic/ED risks, and high ED incidence, respectively. This hierarchy supports individualized therapy, emphasizing monitoring for hyperkalemia and renal function to optimize outcomes in PA's high-risk profile.